Aciparticipa Medical Information For A Better Live

Acute Osteomyelitis: treatment

Acute hematogenous osteomyelitis is caused by a germ that comes from the circulatory system and is almost always caused, as confirmed by positive blood cultures, from Staphylococcus aureus. È It is more common in children. For abundant vascular supply places hardest hit by hematogenous osteomyelitis are the metaphysic of long bones. Therapy proximity to osteomyelitis without generalized vascular insufficiency makes use of adequate drainage, debridement and closure of the gaps. Next surgery should not miss a proper antibiotic coverage.

Osteomyelitis by contiguity with generalized vascular insufficiency should determine the status of vascularization, measuring the skin oxygen tension. The interventions of revascularization, but also the hyperbaric therapy, would facilitate the healing of the areas where the oxygen tension is borderline. Depending on the case the patient can be treated with suppressive antibiotic therapy with surgical debridement with local or radical surgery. Hematogenous osteomyelitis requires a correct antibiotic therapy accompanied, if necessary, by some surgical procedures (adequate drainage, appropriate and thorough debridement, closure of dead space, protection of the wound). Antibiotic therapy should be performed on the basis of the pathogen identified. The patient is subjected to appropriate antimicrobial therapy for a period of 4-6 weeks.

Incoming search terms for the article:

Acute Osteomyelitis: characteristics

The term landlord and myelo refer respectively to the bone and marrow cavity, both involved in the infectious process. Osteomyelitis is in fact due to a bacterial or fungal infection of the bone. Acute osteomyelitis is a very probable hypothesis in patients with localized bone pain, fever or septic state. This infectious disease has a progressive course and results in inflammatory destruction of bone necrosis and information of another bone. The radiographic changes (swelling of soft tissue swelling of the periosteum) can occur several weeks after clinical onset and earlier diagnosis is given to us by bone scintigraphy with technetium and biopsy of suspicious lesions.

We must distinguish acute osteomyelitis contiguity without generalized vascular insufficiency from acute osteomyelitis contiguity with generalized vascular insufficiency. In the first case the organism may spread to the bone through adjacent soft tissue infections, or be injected into the bone from trauma or surgery. The fractures, the surgical reduction with internal fixation of fractures, chronic infections of the soft parts and radiotherapy are the most frequent predisposing conditions of the condition. Bone infection is generally different bacterial strains isolated even if Staphylococcus aureus is the microorganism most frequently found.

Osteomielite by contiguity with generalized vascular insufficiency, most patients suffering from diabetes mellitus or atherosclerotic peripheral vascular disease. The infection usually affects the small bones of the foot, the talus, the calcaneus, distal fibula and tibia. As a result of trauma, even slightly, the soft tissues of the foot (such as an infection unghiale) lead to infection of the bone. The reduced arterial blood supply has traditionally been considered the main predisposing factor.

Incoming search terms for the article: